HIV virus does not cause AIDS Acquired Immune Deficiency Syndrome

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'HIV test a crime, AIDS 'cure' killed a whole generation'

https://www.youtube.com/watch?v=buoGGsch5mM

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HIV = AIDS, Fact or Fraud?
https://topdocumentaryfilms.com/hiv-aids-fact-or-fraud/?fbclid=IwAR1O8Kpl17I08sFxdqU9z4XuyIA4wzU11-fmGBafi7An-XBS8MPE-Oqfd4k

Too many people are making too much money out of it, and money is much stronger than truth. One of the most powerful video documentaries of our time boldly reveals the modern medical-industrial complex’s dire descent into utter corruption.

This feature-length expose explains exactly how the 300-Billion-dollar AID$ fraud began, why HIV can NOT be the cause of AIDS, what the real causes could be, and who manipulates the public's good intentions while poisoning hundreds of thousands with toxic drugs that cause the very disease they are supposed to prevent.

This is a systematic dissection of the HIV/AID$ machine and how they hijacked a program designed to fight a worldwide plight of human suffering and drove it down the road to hell. Yet this program offers hope, inspired by the courage and articulate arguments of a group of growing voices internationally challenging the HIV=AIDS=DEATH hysteria. A MUST SEE for anyone interested in truly understanding the facts about HIV/AID$.

You will meet a number of highly reputable scientists who all agree that HIV doesn't cause AIDS, including Dr. Peter Duesberg, who was the first scientist to map the genetic structure of retroviruses. He is joined by Nobel Prize winners Dr. Kary Mullis and Dr.Walter Gilbert, along with Dr. David Rasnick, an expert in the field of protease inhibitors.


 
Is HIV/AIDS the golden idol of junk science? Judge for yourself. Professionally produced, written and researched, acclaimed by physicians, scientists, journalists and humanitarians internationally, this is the video encyclopedia of HIV/AIDS dissident movement! THE defining documentary on the HIV/AIDS fraud.

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Endogenous retrovirus
https://en.wikipedia.org/wiki/Endogenous_retrovirus?fbclid=IwAR0RAR8ZYKmXUCXer2l2AqyYdbW-vD0V1m9mvK5CigZiWef4wGs5Cet0xm4

Endogenous retroviruses (ERVs) are endogenous viral elements in the genome that closely resemble and can be derived from retroviruses. They are abundant in the genomes of jawed vertebrates, and they comprise up to 5–8% of the human genome (lower estimates of ~1%).[1][2] ERVs are a subclass of a type of gene called a transposon, which can be packaged and moved within the genome to serve a vital role in gene expression and in regulation.[3][4] They are distinguished as retrotransposons, which are Class I elements.[5] Researchers have suggested that retroviruses evolved from a type of transposable gene called a retrotransposon, which includes ERVs; these genes can mutate and instead of moving to another location in the genome they can become exogenous or pathogenic. This means that not all ERVs may have originated as an insertion by a retrovirus but that some may have been the source for the genetic information in the retroviruses they resemble.[6] When integration of viral DNA occurs in the germ-line, it can give rise to an ERV, which can later become fixed in the gene pool of the host population.[1][7]

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Dr Robert Willner Injects "HIV" into himself on TV
https://www.youtube.com/watch?v=tQCKb1JV-4A

December 7th 1994  Hollywood Roosevelt Hotel, Greensboro, N.C.,

Dr Willner (a medical doctor of 40 years experience) an outspoken whistleblower of the AIDS hoax.

infront of a gathering of about 30 alternative-medicine practitioners and several journalists, Willner stuck a needle in the finger of Andres, 27, a Fort Lauderdale student who says he has tested positive for HIV. Then, wincing, the 65-year-old doctor stuck himself.

In 1993, Dr. Willner stunned Spain by inoculating himself with the blood of Pedro Tocino, an HIV positive hemophiliac. This demonstration of devotion to the truth and the Hippocratic Oath he took, nearly 40 years before, was reported on the front page of every major newspaper in Spain. His appearance on Spain's most popular television show envoked a 4 to 1 response by the viewing audience in favor of his position against the "AIDS hypothesis." When asked why he would put his life on the line to make a point, Dr. Willner replied: "I do this to put a stop to the greatest murderous fraud in medical history. By injecting myself with HIV positive blood, I am proving the point as Dr. Walter Reed did to prove the truth about yellow fever. In this way it is my hope to expose the truth about HIV in the interest of all mankind."

He tested negative multiple times.
He died of a Heart attack 4 months later 15th April 1995

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Deadly Deception the Proof That Sex And HIV Absolutely Do Not Cause AIDS
Hardcover – September 1, 1994
by Robert E. Willner

https://www.amazon.com/Deadly-Deception-Proof-Absolutely-Cause/dp/0964231611

It is heartbreaking that the public is completely unaware of the terrible crime against humanity that has been committed by our own governments. Dr Robert Willner was a very brave man who died trying to expose the truth about HIV. The truth is this: HIV is a harmless retrovirus that was shamelessly used by the US government to kill a lot of people with AZT, scare the general public into using condoms ( population control ), and make a hell of a lot of money for the pharmaceutical companies. Read this book and see that George Orwell's 1984 has come to pass after all.

Incidentally, there is a great video from 1994 on Youtube in which Dr. Robert Willner explains the HIV hoax to the press corps and then sticks his own finger with a needle that is potentially HIV infected. He put his money where his mouth is to help average people to see the truth.

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http://www.ygeiaonline.gr/component/k2/item/9237-azt

AZT (αζιδοθυμιδίνη). Η πρώτη φαρμακευτική ουσία που εγκρίθηκε για τη θεραπευτική αντιμετώπιση ασθενών προσβεβλημένων από τον ιό HIV, που είναι υπεύθυνος για το σύνδρομο επίκτητης ανοσολογικής ανεπάρκειας, πιο γνωστό ως AIDS. Η ουσία ονομάζεται εναλλακτικά zidovudine και η εμπορική του ονομασία είναι retrovir. Ανακαλύφθηκε το 1964 ως αντινεοπλασματικό φάρμακο, αλλά εγκαταλείφθηκε γρήγορα λόγω της υψηλής κυτταροτοξικότητάς του. Το 1984 μελέτες έδειξαν δραστικότητα του AZT εναντίον του ιού HIV· αφού πήρε έγκριση από τον αρμόδιο φορέα στις ΗΠΑ, σε χρόνο ρεκόρ άρχισε να συνταγογραφείται σε ασθενείς με AIDS. Το ΑZT είναι αναστολέας της ανάστροφης τρανσκριπτάσης, ενός ενζύμου αναγκαίου για τον πολλαπλασιασμό του ιού HIV. Δρα ξεγελώντας τον ιό, αναγκάζοντάς τον να παράγει ελαττωματική ανάστροφη τρανσκριπτάση και εμποδίζοντας έτσι την αναπαραγωγή του. Είναι ένα από τα λίγα φάρμακα που μπορεί να επιτεθεί στον ιό HIV στο κεντρικό νευρικό σύστημα. Τα βραχυπρόθεσμα αποτελέσματά του στην αύξηση του σωματικού βάρους του ασθενούς και στη μείωση των δευτερογενών (ευκαιριακών) λοιμώξεων επισκιάζονται από την ελάττωση της αποτελεσματικότητάς του με τον χρόνο και τις πιθανές σοβαρές παρενέργειές του. Δεν έχει αποδειχθεί δραστικότητα της ουσίας αυτής σε φορείς του AIDS ώστε να μην εκδηλώσουν τη νόσο, ούτε στη μείωση της μεταδοτικότητας, αν και μελετητές υποστηρίζουν ότι μειώνει την πιθανότητα μετάδοσης από την έγκυο στο έμβρυο. Το AZT είναι το συχνότερα συνταγογραφούμενο φάρμακο για το AIDS, αποτελεί όμως και αντικείμενο μεγάλης αμφισβήτησης λόγω των παρενεργειών του, ανάμεσα στις οποίες κυριαρχεί η κυτταροτοξικότητα που επιφέρει αναιμία, η κοκκιοκυτταροπενία και η γαστρεντερική δυσανεξία. Επίσης, έχουν αναφερθεί υπέρταση, αδιαθεσία και μυϊκή εξασθένιση. Η ταυτόχρονη χορήγηση του AZT με άλλα κατάλληλα φάρμακα μειώνει όχι μόνον τη συχνότητα εμφάνισης και την ένταση των παρενεργειών, αλλά και την πιθανότητα εμφάνισης αντοχής στο φάρμακο λόγω ανάπτυξης μεταλλαγμένων στελεχών του ιού.

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https://positivevoice.gr/205

ΑΝΤΙΡΕΤΡΟΪΚΑ ΦΑΡΜΑΚΑ
Ημερομηνία ανάρτησης: 22 Ιανουαρίου 2010
Η θεραπεία κατά του ιού HIV συνήθως περιλαμβάνει 3 αντιρετροϊκά φάρμακα από τις παρακάτω ομάδες φαρμάκων.
Ο συνδυασμός αντιρετροϊκών φαρμάκων έχει σκοπό την αναστολή της δημιουργίας ιϊκών αντιγράφων, προσπαθώντας να ελαχιστοποιήσει τις πιθανές ανεπιθύμητες ενέργειες καθώς και τον αριθμό των λαμβανομένων δισκίων.
Η Παγκόσμια Εταιρεία AIDS (IAS) συνιστά κατά σειρά προτίμησης έναν από τους εξής αρχικούς θεραπευτικούς συνδυασμούς για την αντιμετώπιση του HIV:

1 NNRTI + 2 NRTI
Atripla = (Stocrin + Truvada)
1 PI + 2 NRTI
Reyataz/norvir + Truvada
Prezista/norvir + Truvada
Telzir/norvir + Truvada
Kaletra + Truvada
Στον παρακάτω πίνακα θα βρείτε στα Ελληνικά, τα φύλλα οδηγιών χρήσης ανά κατηγορία όλων των εγκεκριμένων αντιρετροϊκών φαρμάκων για την αντιμετώπιση της HIV λοίμωξης.

Πολυφάρμακα
Atripla   Atripla (efavirenz + tenofovir + emtricitabine)
Νουκλεοσιδικοί / νουκλεοτιδικοί Αναστολείς ανάστροφης τρανσκριπτάσης (NRTIs)

Combivir   Combivir (zidovudine + lamivudine, AZT + 3TC)
Emtriva   Emtriva (emtricitabine, FTC)
Epivir   Epivir (lamivudine, 3TC)
Epzicom   Kivexa (abacavir + lamivudine, ABC + 3TC)
Retrovir   Retrovir (zidovudine, AZT, ZDV)
Trizivir   Trizivir (abacavir + zidovudine + lamivudine, ABC + AZT + 3TC)
Truvada   Truvada (tenofovir DF + emtricitabine, TDF + FTC)
Videx   Videx & Videx EC (didanosine, ddI)
Viread   Viread (tenofovir disoproxil fumarate, TDF)
Zerit   Zerit (stavudine, d4T)
Ziagen   Ziagen (abacavir, ABC)
Μη Νουκλεοσιδικοί Αναστολείς ανάστροφης τρανσκριπτάσης (NNRTIs)

Intelence   Intelence (etravirine, ETV, TMC-125)
Sustiva   Stocrin (efavirenz, EFV)
Viramune   Viramune (nevirapine, NVP)
Αναστολείς πρωτεάσης (PIs)

Aptivus   Aptivus (tipranavir, TPV)
Crixivan   Crixivan (indinavir, IDV)
Invirase   Invirase (saquinavir, SQV)
Kaletra   Kaletra (Aluvia, lopinavir/ritonavir, LPV/r)
Lexiva   Telzir (fosamprenavir, FPV)
Norvir   Norvir (ritonavir, RTV)
Prezista   Prezista (darunavir, DRV)
Reyataz   Reyataz (atazanavir, ATV)
Viracept   Viracept (nelfinavir, NFV)
Αναστολείς εισόδου-σύντηξης
Fuzeon   Fuzeon (enfuvirtide, ENF, T-20)
maraviroc   Celsentri (maraviroc, UK-427,857)
Αναστολείς ιντεγκράσης
Isentress   Isentress (raltegravir, MK-0518)

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https://ec.europa.eu/health/documents/community-register/2004/200405267918/anx_7918_el.pdf

https://eody.gov.gr/wp-content/uploads/2018/12/antiretroiki-agogi-se-paidia.pdf

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https://www.ncbi.nlm.nih.gov/pubmed/3299090

N Engl J Med. 1987 Jul 23;317(4):192-7.
The toxicity of azidothymidine (AZT) in the treatment of patients with AIDS and AIDS-related complex. A double-blind, placebo-controlled trial.
Richman DD, Fischl MA, Grieco MH, Gottlieb MS, Volberding PA, Laskin OL, Leedom JM, Groopman JE, Mildvan D, Hirsch MS, et al.

Abstract
We conducted a double-blind, placebo-controlled trial of oral azidothymidine (AZT) in 282 patients with the acquired immunodeficiency syndrome (AIDS) or AIDS-related complex. Although significant clinical benefit was documented (N Engl J Med 1987; 317:185-91), serious adverse reactions, particularly bone marrow suppression, were observed. Nausea, myalgia, insomnia, and severe headaches were reported more frequently by recipients of AZT; macrocytosis developed within weeks in most of the AZT group. Anemia with hemoglobin levels below 7.5 g per deciliter developed in 24 percent of AZT recipients and 4 percent of placebo recipients (P less than 0.001). Twenty-one percent of AZT recipients and 4 percent of placebo recipients required multiple red-cell transfusions (P less than 0.001). Neutropenia (less than 500 cells per cubic millimeter) occurred in 16 percent of AZT recipients, as compared with 2 percent of placebo recipients (P less than 0.001). Subjects who entered the study with low CD4 lymphocyte counts, low serum vitamin B12 levels, anemia, or low neutrophil counts were more likely to have hematologic toxic effects. Concurrent use of acetaminophen was also associated with a higher frequency of hematologic toxicity. Although a subset of patients tolerated AZT for an extended period with few toxic effects, the drug should be administered with caution because of its toxicity and the limited experience with it to date.

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https://www.ncbi.nlm.nih.gov/pmc/articles/PMC3858928/

The common causes leading to pancytopenia in patients presenting to tertiary care hospital
Kirpal Das Makheja,1 Bharat Kumar Maheshwari,2 Shafique Arain,3 Suneel Kumar,4 Sangeeta Kumari,5 and Vikash6
Author information Article notes Copyright and License information Disclaimer
This article has been cited by other articles in PMC.

Objective: The objective of this study was to determine the frequency of common causes leading to Pancytopenia in patients presenting to tertiary care hospital at Karachi.

Methods: A total of 62 patients with the diagnosis of Pancytopenia of more than one week duration were enrolled in the study. All patients underwent a detailed medical history and full physical examination followed by blood sampling for the investigations i.e. complete blood count with peripheral film, erythrocyte sedimentation rate (ESR), Malarial parasites (MP), liver function test, Renal function tests, PT and viral profile (HBsAg, Anti-HCV), Ultrasonography of abdomen. All patients underwent bone marrow aspiration and trephine biopsy for reporting and interpretation. Duration of study was six months, from May 2010 to November 2010.

Results: The average age of the patients was 37.76 ± 16.38years. Out of 62 patients, 36 (58%) were male and 26 (42%) were female. Megaloblastic anemia was the commonest cause that was observed in 41.9% cases followed by acute myeloid leukemia 27.4%, aplastic anemia 19.4% and erythroid hyperplasia 11.3%.

Conclusion : This study concluded that most common cause of pancytopenia is Megaloblastic anemia, followed by acute myeloid leukemia and aplastic anemia. Bone marrow examination is a single useful investigation which reveals the underlying cause in patients with pancytopenia.

Key Words: Aplastic Anemia, Bone Marrow Biopsy, Megaloblastic Anemia, Pancytopenia

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False positive results on HIV tests
https://www.aidsmap.com/about-hiv/false-positive-results-hiv-tests

What causes false positive results?

HIV tests are based on the detection of antibodies to HIV. These are proteins produced by the immune system in response to a foreign substance, such as HIV. The main cause of false positive results is that the test has detected antibodies, but they are not antibodies to HIV – they are antibodies to another substance or infection. Tests are not meant to react to other types of antibodies, but it sometimes happens.

There are other reasons why a test may give a false positive result. Depending on the testing device, reading the test result may rely on subjective interpretation. When the result is borderline, experienced staff give more consistently accurate results. A false positive result could also be the result of a sample being mislabelled, mixed up with another person’s, or some other clerical or technical error.

Less commonly, false positive results may occur in people who have recently had a flu vaccine, are taking part in an HIV vaccine study, or have an autoimmune disease (such as lupus).

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https://www.researchgate.net/publication/261948355_Inventing_the_AIDS_Virus

Inventing the AIDS Virus

Foreword P.H. Duesberg. A Critical Analysis of the HIV-T4-cell-AIDS Hypothesis E. Papadopulos-Eleopulos, et al. Factor VIII, HIV and Aids in Haemophiliacs: An Analysis of their Relationship E. Papadopulos-Eleopulos, et al. Foreign-Protein-Mediated Immunodeficiency in Hemophiliacs with and without HIV P.H. Duesberg. Critical Analysis of the Current Views on the Nature of AIDS V.L. Koliadin. Some Mathematical Considerations on HIV and AIDS M. Craddock. HIV as a Surrogate Marker for Drug Use: A Re-analysis of the San Francisco Men's Health Study B.J. Ellison, et al. A Critical Appraisal of the Vancouver Men's Study Does it Refute the Drugs/AIDS Hypothesis? M. Craddock. Duesberg and the Right of Reply According to Maddox-Nature P.H. Duesberg, H. Bialy. HIV: Science by Press Conference M. Craddock. AZT Toxicity and AIDS Prophylaxis: Is AZT Beneficial for HIV+ Asymptomatic Persons with 500 or More T4 Cells per Cubic Millimeter? M.D. Zaretsky. The Toxicity of Azidothymidine (AZT) on Human and Animal Cells in Culture at Concentrations Used for Antiviral Therapy D.T. Chui, P.H. Duesberg. Measuring Inhalant Nitrite Exposure in Gay Men: Implications for Elucidating the Etiology of AIDS-Related Kaposi's Sarcoma H.W. Haverkos, D.P. Drotman. A Hypothetical Disease of the Immune System That May Bear Some Relation to the Acquired Immune Deficiency Syndrome K.B. Mullis. The Epidemiology and Transmission of AIDS: A Hypothesis Linking Behavioural and Biological Determinants to Time, Person and Place G.T. Stewart. Five Myths About AIDS that have Misdirected Research and Treatment R.S. Root-Bernstein. Semen Alloantigens and Lymphocytotoxic Antibodies in AIDS and ICL R.S. Root-Bernstein, S. Hobbs de Witt. AIDS and Good Theory-Making S. Harris. How Much Longer Can We Afford the AIDS Virus Monopoly? P.H. Duesberg. HIV and AIDS: Have we Been Misled? Questions of Scientific and Journalistic Responsibility S. Lang. To Fund or Not to Fund, That is the Question: Proposed Experiments on the Drug--AIDS Hypothesis To Inform or Not to Inform, That is Another Question S. Lang. HIV Symposium at AAAS Conference J. Lauritsen. AIDS and Poppers T. Bethell. NIDA Meeting Calls for Research into the Poppers--Kaposi's Sarcoma Connection J. Lauritsen. The Thinking Problem in HIV-Science P.E. Johnson. The Incidence Quagmire J. Lauritsen. The HIV Test C. Farber. Cry, Beloved Country: How Africa Became the Victim of a Non-Existent Epidemic of HIV/AIDS N. Hodgkinson.

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https://www.researchgate.net/publication/303473089_Some_mathematical_considerations_on_HIV_and_AIDS

Some mathematical considerations on HIV and AIDS
Chapter · January 1996 with 138 Reads 

It is commonly accepted that HIV is both necessary and sufficient to cause the immunodeficiency and multiple diseases seen in patients diagnosed with AIDS. In other words it is accepted that HIV is the cause of AIDS. Upon this basis public health decisions in all Western countries regarding AIDS are made. However, many scientists now question the role of the virus (Root-Bernstein, 1993). Questions that have arisen about the virus include whether or not it is present in sufficient quantities to cause disease and whether or not AIDS is infectious. The former question has been applied to by new studies using the Polymerase Chain Reaction (PCR) technique that claim to detect very large quantities of virus in HIV+ patients at all stages of disease progression. I will examine these studies and show that they do not truly answer the criticisms that have been levelled. They in fact give rise to more questions than they answer. Predictions that one can make from them contradict the observed pattern of the disease. I will also argue that data based upon the so called Quantitative Competitive PCR need to be treated with caution.

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'HIV test a crime, AIDS 'cure' killed a whole generation'
https://www.youtube.com/watch?v=buoGGsch5mM&t=10s

Thousands of experts, scientists and politicians from around the globe have gathered in Vienna to discuss HIV/AIDS prevention. But the convention has also attracted sceptics who oppose mainstream beliefs surrounding the deadly virus. To discuss the alternative HIV/AIDS conference RT talks to Joan Shenton, founder of the Immunity Resource Foundation.

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https://www.amazon.com/HIV-AIDS-Greatest-Century-profitable-business-ebook/dp/B07CRLPRPH


HIV-AIDS SCAM EXPOSED 8th APRIL PART - I
https://www.youtube.com/watch?v=fNIQ1s_6sO8


http://biswaroop.com/HIV

HIV-AIDS : Greatest Lie of 21 Century and the most profitable business Kindle Edition
by Dr. Biswaroop Roy Chowdhury  (Author)

This book touches the most sensitive subject of the modern world. The truth given in the book is going to shake the faith of humanity on modern medicine forever. To avoid the confusion and misunderstanding about the reality of HIV/AIDS hypothesis and keeping in mind that the subject is already too complicated with lot of stigma and taboo attached around it, the book is divided into two sections. Section Ist attempts to explain the whole story in a plain language, while the section Ilnd is in a format more acceptable to the scientific community. However, the examples in both the sections are mutually exclusive. With this, I hope the book will be able to relieve the mankind of the economic, social and psychological burden of HIV/ AIDS...forever.

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https://www.researchgate.net/publication/20510863_Human_immunodeficiency_virus_and_acquired_immunodeficiency_syndrome_Correlation_but_no_causation

Human immunodeficiency virus and acquired immunodeficiency syndrome: Correlation but no causation

AIDS is an acquired immunodeficiency syndrome defined by a severe depletion of T cells and over 20 conventional degenerative and neoplastic diseases. In the U.S. and Europe, AIDS correlates to 95% with risk factors, such as about 8 years of promiscuous male homosexuality, intravenous drug use, or hemophilia. Since AIDS also correlates with antibody to a retrovirus, confirmed in about 40% of American cases, it has been hypothesized that this virus causes AIDS by killing T cells. Consequently, the virus was termed human immunodeficiency virus (HIV), and antibody to HIV became part of the definition of AIDS. The hypothesis that HIV causes AIDS is examined in terms of Koch's postulates and epidemiological, biochemical, genetic, and evolutionary conditions of viral pathology. HIV does not fulfill Koch's postulates: (i) free virus is not detectable in most cases of AIDS; (ii) virus can only be isolated by reactivating virus in vitro from a few latently infected lymphocytes among millions of uninfected ones; (iii) pure HIV does not cause AIDS upon experimental infection of chimpanzees or accidental infection of healthy humans. Further, HIV violates classical conditions of viral pathology. (i) Epidemiological surveys indicate that the annual incidence of AIDS among antibody-positive persons varies from nearly 0 to over 10%, depending critically on nonviral risk factors. (ii) HIV is expressed in less than or equal to 1 of every 10(4) T cells it supposedly kills in AIDS, whereas about 5% of all T cells are regenerated during the 2 days it takes the virus to infect a cell. (iii) If HIV were the cause of AIDS, it would be the first virus to cause a disease only after the onset of antiviral immunity, as detected by a positive "AIDS test." (iv) AIDS follows the onset of antiviral immunity only after long and unpredictable asymptomatic intervals averaging 8 years, although HIV replicates within 1 to 2 days and induces immunity within 1 to 2 months. (v) HIV supposedly causes AIDS by killing T cells, although retroviruses can only replicate in viable cells. In fact, infected T cells grown in culture continue to divide. (vi) HIV is isogenic with all other retroviruses and does not express a late, AIDS-specific gene. (vii) If HIV were to cause AIDS, it would have a paradoxical, country-specific pathology, causing over 90% Pneumocystis pneumonia and Kaposi sarcoma in the U.S. but over 90% slim disease, fever, and diarrhea in Africa.(viii) It is highly improbable that within the last few years two viruses (HIV-1 and HIV-2) that are only 40% sequence-related would have evolved that could both cause the newly defined syndrome AIDS. Also, viruses are improbable that kill their only natural host with efficiencies of 50-100%, as is claimed for HIVs. It is concluded that HIV is not sufficient for AIDS and that it may not even be necessary for AIDS because its activity is just as low in symptomatic carriers as in asymptomatic carriers. The correlation between antibody to HIV and AIDS does not prove causation, because otherwise indistinguishable diseases are now set apart only on the basis of this antibody. I propose that AIDS is not a contagious syndrome caused by one conventional virus or microbe. No such virus or microbe would require almost a decade to cause primary disease, nor could it cause the diverse collection of AIDS diseases. Neither would its host range be as selective as that of AIDS, nor could it survive if it were as inefficiently transmitted as AIDS. Since AIDS is defined by new combinations of conventional diseases, it may be caused by new combinations of conventional pathogens, including acute viral or microbial infections and chronic drug use and malnutrition. The long and unpredictable intervals between infection with HIV and AIDS would then reflect the thresholds for these pathogenic factors to cause AIDS diseases, instead of an unlikely mechanism of HIV pathogenesis.

[Rose]

https://www.researchgate.net/publication/20510863_Human_immunodeficiency_virus_and_acquired_immunodeficiency_syndrome_Correlation_but_no_causation

AIDS is an acquired immunodeficiency syndrome defined by a severe depletion of T cells and over 20 conventional degenerative and neoplastic diseases. In the U.S. and Europe, AIDS correlates to 95% with risk factors, such as about 8 years of promiscuous male homosexuality, intravenous drug use, or hemophilia. Since AIDS also correlates with antibody to a retrovirus, confirmed in about 40% of American cases, it has been hypothesized that this virus causes AIDS by killing T cells. Consequently, the virus was termed human immunodeficiency virus (HIV), and antibody to HIV became part of the definition of AIDS. The hypothesis that HIV causes AIDS is examined in terms of Koch's postulates and epidemiological, biochemical, genetic, and evolutionary conditions of viral pathology. HIV does not fulfill Koch's postulates: (i) free virus is not detectable in most cases of AIDS; (ii) virus can only be isolated by reactivating virus in vitro from a few latently infected lymphocytes among millions of uninfected ones; (iii) pure HIV does not cause AIDS upon experimental infection of chimpanzees or accidental infection of healthy humans. Further, HIV violates classical conditions of viral pathology. (i) Epidemiological surveys indicate that the annual incidence of AIDS among antibody-positive persons varies from nearly 0 to over 10%, depending critically on nonviral risk factors. (ii) HIV is expressed in less than or equal to 1 of every 10(4) T cells it supposedly kills in AIDS, whereas about 5% of all T cells are regenerated during the 2 days it takes the virus to infect a cell. (iii) If HIV were the cause of AIDS, it would be the first virus to cause a disease only after the onset of antiviral immunity, as detected by a positive "AIDS test." (iv) AIDS follows the onset of antiviral immunity only after long and unpredictable asymptomatic intervals averaging 8 years, although HIV replicates within 1 to 2 days and induces immunity within 1 to 2 months. (v) HIV supposedly causes AIDS by killing T cells, although retroviruses can only replicate in viable cells. In fact, infected T cells grown in culture continue to divide. (vi) HIV is isogenic with all other retroviruses and does not express a late, AIDS-specific gene. (vii) If HIV were to cause AIDS, it would have a paradoxical, country-specific pathology, causing over 90% Pneumocystis pneumonia and Kaposi sarcoma in the U.S. but over 90% slim disease, fever, and diarrhea in Africa.(viii) It is highly improbable that within the last few years two viruses (HIV-1 and HIV-2) that are only 40% sequence-related would have evolved that could both cause the newly defined syndrome AIDS. Also, viruses are improbable that kill their only natural host with efficiencies of 50-100%, as is claimed for HIVs. It is concluded that HIV is not sufficient for AIDS and that it may not even be necessary for AIDS because its activity is just as low in symptomatic carriers as in asymptomatic carriers. The correlation between antibody to HIV and AIDS does not prove causation, because otherwise indistinguishable diseases are now set apart only on the basis of this antibody. I propose that AIDS is not a contagious syndrome caused by one conventional virus or microbe. No such virus or microbe would require almost a decade to cause primary disease, nor could it cause the diverse collection of AIDS diseases. Neither would its host range be as selective as that of AIDS, nor could it survive if it were as inefficiently transmitted as AIDS. Since AIDS is defined by new combinations of conventional diseases, it may be caused by new combinations of conventional pathogens, including acute viral or microbial infections and chronic drug use and malnutrition. The long and unpredictable intervals between infection with HIV and AIDS would then reflect the thresholds for these pathogenic factors to cause AIDS diseases, instead of an unlikely mechanism of HIV pathogenesis.