Αποστολέας Θέμα: HIV virus does not cause AIDS Acquired Immune Deficiency Syndrome  (Αναγνώστηκε 483 φορές)

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Απ: HIV virus does not cause AIDS Acquired Immune Deficiency Syndrome
« Απάντηση #30 στις: Οκτώβριος 10, 2019, 02:02:12 »
ΒΙΒΛΙΟΓΡΑΦΙΑ:
1. A. Hässig, H. Kremer, Liang Wen-Xi and K. Stampfli : Pathogenesis of human suppression in hypercatabolic diseases:AIDS, septicaemia, toxic shock syndrome and protein calorie malnutrition (Continuum vol.4 no.6 June/July 1997

2. -Prof. Alfred Hässig, Prof. Liang Wen-Xi and Dr. Kurt Stampfli: Reappraisal of the depletion of circulating CD4+ lymphocytes in HIV-carriers in transition to AIDS ( Continuum vol.3 no.5 Jan./Feb. 1996)
3.   -DID DR. GALLO AND HIS COLLEAGUES MANIPULATE THE «AIDS-TEST» TO ORDER? «The hunt for the virus» 1 has degenerated into «clean torture with fatal result» 2By Heinrich Kremer,Continuum Summer 1998

4.     INTERVIEW STEFAN LANKA Challenging BOTH Mainstream and Alternative AIDS Views By Mark Gabrish Conlan Zenger’s Dec. 1998
5.    -Eleni Papadopulos-Eleopulos (1) Valendar F.Turner (2) John M. Papadimitriou (3) David Causer (1) Department of Medical Physics, (2) Department of Emergency Medicine, Royal Perth Hospital, Perth, Western Australia; (3) Department of Pathology, University of Western Australia. THE ISOLATION OF HIV—HAS IT REALLY BEEN ACHIEVED? THE CASE AGAINST( Continuum
Vol.4 No.3 Sept./Oct. 1996)
6.  E. Papadopulos-Eleopulos, ‘Reappraisal of AIDS; Is the oxidation induced by risk factors the primary cause?’ Medical Hypothesis 1988, vol. 25 pp.151-162.
7. E. Papadopulos-Eleopulos, B. Hedland-Thomas, D.A. Causer & A.P. Dufty, ‘An alternative explanation for the radiosensitization of AIDS patients’ (letter) Int. Jour. Radiation Oncology Biol. Physics. 1989, vol.17 pp.695-697.
8.  V.F. Turner, ‘Reducing agents and AIDS – why are we waiting?’ (letter) The Medical Journal of Australia 15 Oct. 1990, vol.153 p.502.
9.  E. Papadopulos-Eleopulos, B. Hedland-Thomas, D.A. Causer, V.F. Turner & J.M. Papadimitriou, ‘Changes in thiols and glutamate as consequence of SIV infection’ (letter) The Lancet 19 Oct. 1991, vol.338 p.1013.
10.  E. Papadopulos-Eleopulos, V.F. Turner & J.M. Papadimitriou, ‘Kaposie sarcoma and HIV’ Medical Hypothesis 1992, vol.39 pp.22-29.
11.  E. Papadopulos-Eleopulos, V.F. Turner & J.M. Papadimitriou, ‘Oxidative stress, HIV and AIDS’ Research in Immunology 1992, vol.143 pp.145-148.
12. E. Papadopulos-Eleopulos, V.F. Turner & J.M. Papadimitriou, ‘Is a western blot proof of HIV infection?’ Bio/Technology 11 June 1993, vol.11 pp.696-707.
13.  E. Papadopulos-Eleopulos, V.F. Turner & J.M. Papadimitriou, ‘Has Gallo proven the role of HIV in AIDS?’ Emergency Medicine 1993, vol.5 pp.71-147.
14.  V. F. Turner, ‘HIV western blot test’ (letter) The Medical Journal of Australia, 20 June 1994, vol.160 pp.807,808.
15. E. Papadopulos-Eleopulos, V.F. Turner & J.M. Papadimitriou, D. Causer, B. Hedland-Thomas and B. Page, ‘A critical analysis of the HIV-T4-cell-AIDS hypothesis’ Genetica March 1995, vol.95 pp.5-24.
16.  E. Papadopulos-Eleopulos, V.F. Turner, J.M. Papadimitriou & D. Causer, ‘Factor VIII, HIV and AIDS in haemophiliacs: an analysis of their relationship’ Genetica March 1995, vol.95 pp.25-50.
17.  E. Papadopoulos-Eleopulos, V.F. Turner, J.M. Papadimitriou & H. Bialy, ‘AIDS in Africa: Distinguishing fact and fiction’ World Journal of Microbiology & Biotechnology March 1995, vol.11 pp.135-143
18.  -E. Papadopoulos-Eleopulos, V.F. Turner, J.M. Papadimitriou, G. Stewart & D. Causer ‘HIV Antibodies: Further questions and a plea for clarification’ Current Medical Research and Opinion 1997, vol.13 pp.627-634.
19.  -E. Papadopoulos-Eleopulos, V.F. Turner, J. M. Papadimitriou, D. Causer & B. Page ‘HIV Antibody Tests and Viral Load – More Unanswered Questions and a Further Plea for Clarification’ Current Medical Research and Opinion 1998, vol.14 pp.185-186.
20.  15 YEARS OF AIDS  The continuous failure in the prevention and treatment of AIDS is rooted in the misinterpretation of an inflammatory auto immune process as a lethal, viral venereal disease  By A. Hässig, H. Kremer, S. Lanka, W-X Liang, K. Stampfli (www.virusmyth.com)
21.  -HIV; REALITY OR ARTEFACT? By Stefan Lanka ,Continuum April/May 1995

22. -AIDS: DEATH BY PRESCRIPTION By Heinrich Kremer, Stefan Lanka & Alfred Hässig Continuum, July/Aug. 1996
23. * Δρ Bernard Forscher, πρώην εκδότης των Πρακτικών  της Εθνικής Ακαδημίας των Επιστημών των ΗΠΑ : «Η υπόθεση HIV είναι της ίδιας τάξης  με τη θεωρία του » κακού αέρα «για την ελονοσία και τη θεωρία της » βακτηριακής μόλυνσης »  του beriberi και της  pellagra [ που προκαλούνται από τις θρεπτικές ανεπάρκειες ]. Είναι μια αγυρτεία  που έγινε μιά εξαπάτηση»  (Sunday Times (London) 3 April 1994)
-A. HASSIG, LIANG WEN-XI AND K. STAMPFLI :Stress-induced suppression of the cellular immune reactions.
A contribution on the neuroendocrine control of the immune system., Medical Hypothesis (19964 6: 551-555)

24.  -Dr. Stefan Lanka Exposes The «Viral Fraud»: Pictures of «Isolated Viruses» Debunked

25. -David Rasnick -INHIBITORS OF HIV PROTEASE USELESS AGAINST AIDS BECAUSE HIV DOESN’T CAUSE AIDS Reapprasing AIDS August 1996.

26.Etienne de Harven: RETROVIRUSES -The Recollections of an Electron Microscopist, Reappraising AIDS Nov./Dec. 1998
27.Etienne de Harven: REMARKS ON METHODS FOR RETROVIRAL ISOLATION, Continuum Spring 1998

Χρήσιμες Διευθύνσεις:
* Eleni Papadopulos-Eleopulos
Department of Medical Physics
Royal Perth Hospital
Perth, Western Australia Voice int + 618 92243221
Fax int + 618 92243511
Email: vturner@cyllene.uwa.edu.au
* Study Group for AIDS therapy
c/o Felix A. de Fries
Eglistr. 7 CH-8004 Zürich
Tel./Fax: 0041 1 401 34 24
E-mail: felix.defries@bluewin
Continuum, July/Aug. 1996 Dietetic advice for immunodeficiencyContinuum volume 5, number 5 – mid-winter 1999
* Siro Passi and Chiara De Luca
Cell Aging Center, Istituto Dermopatico dell’Immacolata (IDI) Rome, Italy .

*Prof. Etienne de Harven, « Le Mas Pitou », 2879 Route de Grasse, 06530 Saint Cézaire sur Siagne, France
E-mail <pitou.deharven@wanadoo.fr
*Professor Peter H. Duesberg, Ph.D.
Department of Molecular & Cell Biology
c/o Stanley/Donner Administrative Services Unit
229 Stanley Hall #3206 University of California at Berkeley
Berkeley, CA 94720-3206 Email: duesberg@uclink4.berkeley.edu
Fax: (510) 643-6455
*Prof. LIANG Wen-Xi
Dr. K. STAMPFLI
Study Group Nutrition and Immunity
Elisabethenstr. 51
CH-3014 Bern
Switzerland
*Dr. H. KREMER
Metzendorfer Weg 36
D-21224 Rosengarten-Tštensen b. Hamburg
Germany
*Dr. S. LANKA Im Dreieck 8
D-44143 Dortmund
Germany (a-mail: lanka@free.de)
*HEAL-TORONTO-CANADA
TORONTO tel/fax:(416) 406-HEAL

*Roberto Giraldo:  E-mail: RobGiraldo@aol.com

Στό Ιντερνετ :
Αγγλικά:

http://www.the virus myth.net
http://www.sumeria
http://www.the Perth group
http://www.healtoronto.com
http://www.robertogiraldo.com
http://www.duesberg.com
http://www.angelfire .com/ar/dthcamp
http://www.sickof doctors
ww w.Actup
http://www.alive and well
Ιταλικά:
http://www.macrolibrarsi therapie dott cremer
http://www.controinformationeaids
Γαλλικά:
http://www.perso.wanadoo.fr
http://www.HEAL France
Γερμανικά:
http://www.aids-info.net
http://www.HEALBerlin
http://www.aids-kritik
www. mythos-hiv.de
http://www.Raum undZeit
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Απ: HIV virus does not cause AIDS Acquired Immune Deficiency Syndrome
« Απάντηση #31 στις: Οκτώβριος 10, 2019, 17:09:22 »
The Perth Group - Why HIV has never been discovered
https://www.youtube.com/watch?v=NiwM5K0kJcI

« Τελευταία τροποποίηση: Οκτώβριος 10, 2019, 22:39:41 από Rose »
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Απ: HIV virus does not cause AIDS Acquired Immune Deficiency Syndrome
« Απάντηση #32 στις: Οκτώβριος 10, 2019, 17:14:48 »
http://www.theperthgroup.com/

The view of The Perth Group is that the HIV/AIDS experts have not proven:
1.   The existence of a unique, exogenously acquired retrovirus, HIV.
2.   The "HIV" antibody tests are specific for "HIV" infection.
3.   The HIV theory of AIDS, that is, that HIV causes acquired immune deficiency (destruction of T4 lymphocytes=AID) or that AID leads to the development of the clinical syndrome AIDS.
4.   The "HIV genome", (RNA or DNA) originates in a unique, exogenously acquired infectious retroviral particle.
5.   HIV/AIDS is infectious, either by blood, blood products or sexual intercourse.
6.   Mother to child transmission of a retrovirus HIV or its inhibition with AZT or nevirapine.


The Perth Group has argued:
1.   The impossibility of haemophiliacs acquiring HIV following factor VIII infusions.
2.   That AIDS and all the phenomena inferred as "HIV" are induced by changes in cellular redox brought about by the oxidative nature of substances and exposures common to all the AIDS risk groups and to the cells used in the "culture" and "isolation" of "HIV".
3.   That AIDS will not spread outside the original risk groups.
4.   That the cessation of exposure to oxidants and/or use of anti-oxidants will improve the outcome of AIDS patients.
5.   That the pharmacological data prove AZT cannot kill "HIV" and AZT is toxic to all cells and may cause some cases of AIDS.
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Απ: HIV virus does not cause AIDS Acquired Immune Deficiency Syndrome
« Απάντηση #33 στις: Οκτώβριος 10, 2019, 17:22:52 »
Evidence that HIV antibody tests are not specific
https://www.youtube.com/watch?v=nS-ytZfNWXw

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Απ: HIV virus does not cause AIDS Acquired Immune Deficiency Syndrome
« Απάντηση #34 στις: Οκτώβριος 10, 2019, 21:55:26 »
Positive Hell - International Cut
https://www.youtube.com/watch?v=GekHvqIFKgA

__________

The film "Positive Hell" is to be screened at the Portobello Film Festival on Saturday 10th September at around 8 pm. The director, Andi Reiss and Joan Shenton will be there from 7.

Venue: KPH (Kensington Park Hotel) 130 Ladbroke Grove, London W10 6HJ,
https://www.facebook.com/HouseOfNumbers/photos/rpp.40491054861/10154052779184862/?type=3&theater
« Τελευταία τροποποίηση: Οκτώβριος 10, 2019, 22:06:04 από Rose »
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Απ: HIV virus does not cause AIDS Acquired Immune Deficiency Syndrome
« Απάντηση #35 στις: Οκτώβριος 10, 2019, 22:03:50 »
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Απ: HIV virus does not cause AIDS Acquired Immune Deficiency Syndrome
« Απάντηση #36 στις: Οκτώβριος 10, 2019, 22:43:18 »
Endogenous retrovirus
https://en.wikipedia.org/wiki/Endogenous_retrovirus?fbclid=IwAR2FvS4IG98l0XwMt7e4L2b5nXa009mkoPmvlwQFL-yTFkMKj3mg_3ipMz0


Endogenous retroviruses (ERVs) are endogenous viral elements in the genome that closely resemble and can be derived from retroviruses. They are abundant in the genomes of jawed vertebrates, and they comprise up to 5–8% of the human genome (lower estimates of ~1%).[1][2] ERVs are a subclass of a type of gene called a transposon, which can be packaged and moved within the genome to serve a vital role in gene expression and in regulation.[3][4] They are distinguished as retrotransposons, which are Class I elements.[5] Researchers have suggested that retroviruses evolved from a type of transposable gene called a retrotransposon, which includes ERVs; these genes can mutate and instead of moving to another location in the genome they can become exogenous or pathogenic. This means that not all ERVs may have originated as an insertion by a retrovirus but that some may have been the source for the genetic information in the retroviruses they resemble.[6] When integration of viral DNA occurs in the germ-line, it can give rise to an ERV, which can later become fixed in the gene pool of the host population.[1][7]
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Απ: HIV virus does not cause AIDS Acquired Immune Deficiency Syndrome
« Απάντηση #37 στις: Οκτώβριος 10, 2019, 22:59:37 »
https://www.pnas.org/content/pnas/113/33/9155.full.pdf
Extracellular vesicles and viruses: Are they close relatives?
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Απ: HIV virus does not cause AIDS Acquired Immune Deficiency Syndrome
« Απάντηση #38 στις: Οκτώβριος 10, 2019, 23:01:20 »
http://www.theperthgroup.com/HIV/ArakelyanNatureSciRep2017.pdf

Extracellular Vesicles Carry HIV
Env and Facilitate Hiv Infection of
Human Lymphoid Tissue

Anush Arakelyan, Wendy Fitzgerald, Sonia Zicari, Christophe Vanpouille & Leonid Margolis
Cells productively infected with HIV-1 release virions along with extracellular vesicles (EVs) whose
biogenesis, size, and physical properties resemble those of retroviruses. Here, we found that a
significant number of EVs (exosomes) released by HIV-1 infected cells carry gp120 (Env), a viral protein
that mediates virus attachment and fusion to target cells, and also facilitates HIV infection in various
indirect ways. Depletion of viral preparations of EVs, in particular of those that carry gp120, decreases
viral infection of human lymphoid tissue ex vivo. Thus, EVs that carry Env identified in our work seem to
facilitate HIV infection and therefore may constitute a new therapeutic target for antiviral strategy.
It is well established that various cells in vivo and in vitro release extracellular vesicles (EVs) of various size and
biogenesis1
. Many of these vesicles (exosomes) are of the same size as retroviruses, in particular HIV, and are
generated inside the cells along the pathways similar to these viruses2, 3
. Also, these EVs may incorporate proteins
that are common to viruses (e.g., tetraspanins) as well as viral genetic material4, 5
.
Until recently EVs were considered to be “cell dust” but now EVs, in particular the small ones (less than
300nm), are widely studied as a system of cell-cell communication that changes the status of the cells they interact
with6, 7
. EVs seem to affect viral infection8–12, although, the data on the actual effects of EVs on viral infection are
controversial and the mechanisms of these effects remain to be investigated.
Analysis of EVs generated by infected cells as well as the effects of EVs on viral infection are complicated by
the fact that it is almost impossible to separate them from virions in particular from HIV because of the similarities in size and physical properties. Therefore, any HIV preparation is in fact a mixture of HIV virions and EVs.
Here, we overcame some of these problems by segregating EVs through CD45 and/or acetylcholinesterase
(AChE), two proteins that are not incorporated into HIV membranes13–15 and thus can be used to distinguish
EVs from HIV virions. Using our nanotechnology “flow virometry”16, we found that a significant number of EVs
generated in HIV-infected cells carry HIV Env, thus being indistinguishable from “defective” viruses. These EVs
facilitate viral infection in human lymphoid tissue ex vivo, a system that reflects many aspects of HIV infection of
lymphoid tissue in vivo where the critical events of HIV pathogenesis occur17, 18.
Results
EVs released by HIV-infected cells carry Env. In these experiments we distinguished EVs from HIV
virions by the presence of either CD45 or AChE, which are not incorporated into virions in the course of their
biogenesis13–15. We used CD81, a tetraspanin that is incorporated in both HIV and EVs4
, to fish out both entities
for further analysis of their surface antigens. We added magnetic nanoparticles (MNPs) coupled to anti-CD81
antibodies to the preparation of HIVSF162, a prototypical CCR5-tropic HIV produced by PBMC. Then, we stained
the preparation for gp120 with 2G12 fluorescently labeled antibodies, which bind to clusters of high mannose–
type glycans on the outer domain of gp12019, 20 recognizing both monomeric and trimeric Envs, and for CD45
using specific fluorescently labeled antibodies. The results of these experiments are presented in Fig. 1. Almost all
HIV (CD45 negative) particles, as expected, carry gp120. However, unexpectedly, a significant fraction of events
(on average 52.6±5.7% (n=5)) positive for gp120 carry CD45, thus presumably representing EVs (Fig. 1B). As a
control for specificity of gp120 staining, we used EVs released by uninfected PBMC (Fig. 1C).
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Απ: HIV virus does not cause AIDS Acquired Immune Deficiency Syndrome
« Απάντηση #39 στις: Οκτώβριος 10, 2019, 23:05:37 »
http://www.theperthgroup.com/OTHER/nihantibodiesshort.html

NIH / ANTIBODIES

This document was submitted by the Perth Group (www.theperthgroup.com) as part of the Internet debate that took place as a preamble to the Presidential AIDS Advisory Panel meeting held in Johannesburg in July 2000.

In “The Evidence That HIV Causes AIDS” (http://www.niaid.nih.gov/factsheets/evidhiv.htm)  (https://aidsinfo.nih.gov/news/528/the-evidence-that-hiv-causes-aids) one reads:  “Nearly everybody with AIDS has antibodies to HIV…numerous studies from around the world show that virtually all AIDS patients are HIV-seropositive; that is they carry antibodies that indicate HIV infection”.  The relationship between a positive antibody test and AIDS is said to prove that HIV is the cause of AIDS.

There is no doubt that many, if not all, AIDS patients, at least in the USA, Europe and Australia, have a positive antibody test.  However, there is no agreement as to whether these tests “indicate HIV infection".   For example, the packet insert for the Axsym system (HIV-1/HIV-2) manufactured by Abbott Laboratories includes the words “At present there is no recognized standard for establishing the presence or absence of HIV-1 antibody in human blood”.1  This contradicts the above-mentioned NIH document which reads:

“MYTH:  HIV antibody testing is unreliable.

FACT:  Diagnosis of infection using antibody testing is one of the best-established concepts in medicine.  HIV antibody tests exceed the performance of most other infectious disease tests…Current HIV antibody tests have sensitivity and specificity in excess of 98% and are therefore extremely reliable”.2, 3

COMMENTARY

It is incomprehensible how a body of scientists at the National Institutes for Health in the US could present both sides of a scientific debate as a series of "MYTHS" and "FACTS".  Especially without providing the names of scientists who hold the opposing view or any citations to enable the reader to investigate the matter himself.  The only conclusion one can make from this behaviour is that the NIH does not want their readers to learn the full story.

Here we examine one very important "FACT" and leave it up to the reader to make his own judgement as to whether or not it is a “MYTH”.

FACT: THERE IS NO EVIDENCE A RETROVIRUS HAS BEEN ISOLATED FROM THE TISSUES OF AIDS PATIENTS.  HENCE THERE IS NO GOLD STANDARD FOR ANTIBODY TESTING FOR "HIV" INFECTION AND NO PROOF A RETROVIRUS CAUSES AIDS

To prove the specificity of an antibody test or any antibody antigen reaction, one must:

(i)         perform the test in hundreds, if not thousands, of individuals who are assumed to be infected;

(ii)        perform the test in a control group consisting of at least an equal number of individuals who are thought not to be infected, but who are sick;

(iii)       using the same samples prove the existence of HIV by a test independent of the antigen-antibody reaction, that is by using a gold standard for the reaction.

The only gold standard for the HIV antibody test is HIV itself, that is HIV isolation (purification).  At present no such proof exists.4, 5  Nowhere in the cited WHO 1998 reference can one find a gold standard being used to prove the specificity of the antibody test.  All one can find there, as the title indicates, “Comparative Evaluation of the Operational Characteristics of Commercially Available Assays”, is a comparison between 34 HIV test kits against “a panel of 595 human sera (prevalence 33.6% for HIV-1 and 10 % for HIV-2), of which 192 were from Africa, 99 from Asia, 206 from Europe and 98 from South America.  The panel included 332 HIV negative specimens and 203 sera positive for HIV-1 and 60 positive for HIV-2 specimens. In addition the sensitivity of the HIV test kits is assessed on 8 seroconversion panels from Boston Biomedica (BBI)”.   In fact, they did not even use as a gold standard what is at present considered to represent HIV isolation.

Currently, the reaction between an antibody directed against Montagnier’s p24 and antigens in cultures is considered proof for HIV isolation.  Firstly, a reaction between an antibody and an antigen cannot be considered proof for isolation of a retrovirus.  Secondly, the reaction is totally non specific.  In 1992, Jorg Shupbach, the principle author of one of the first four 1984 papers published by Gallo's group on HIV isolation, reported that the whole blood cultures of 49/60 (82%) of "presumably uninfected but serologically indeterminate individuals and 5/5 seronegative blood donors were found positive for p24".6  The non-specificity of the p24 antigen test is so obvious that it is accepted by no less an authority on HIV testing than Philip Mortimer and his colleagues from the UK Public Health Laboratory Service, "Experience has shown that neither HIV culture nor tests for p24 antigen are of much value in diagnostic testing. They may be insensitive and/or non-specific".7  Thirdly, since this reaction is an antibody-antigen reaction itself, it cannot be used as a gold standard for the antibody test.  Even if one uses this reaction as a gold standard for the antibody test, then the WHO data shows the specificity of the antibody test to be very low indeed.  In a large WHO study published in 1994, between 1992-93, 224 specimens were collected in Brazil, Rwanda, Thailand and Uganda from asymptomatic "HIV positive" individuals.  Serostatus was first confirmed in the country of origin and then at the "centralized laboratories responsible for confirming serology, virus isolation, virus expression, and distribution of reagents (George-Speyer-Hans Chemotherapentisches Forschunginstitut (GSH) in Frankfurt, Germany; National Institute for Biological Standards and Control (NIBSC) in London, United Kingdom,; and DAIDS/NIAID in Bethesda, Maryland, United States".  In this WHO study, "of a total of 224 virus cultures, 83 were positive (Isolation rate=37%)".8

As in the WHO reference, in Sloand et al3 no data is presented to prove the specificity of the HIV antibody tests.  It is only stated:  “Antibodies to HIV-1 proteins, which develop during the course of infection, include antibodies to viral core antigen (p24) and antibodies to viral envelope proteins (gp120 and gp41).  Antibodies to HIV-1 polymerase (p55) develop later, if at all.  The most widely used test, the enzyme-linked immunosorbent assay (ELISA), is used in conjunction with a confirmatory test, the Western blot…Although there is variability depending on the test kit used, up to 70% of the initially positive ELISA results are not confirmed by the second ELISA.  Samples that are repeatedly reactive by ELISA must then be confirmed positive by a Western blot or equivalent test.  This procedure enables separation by electrophoresis of individual viral proteins, such as viral core (p24, p55 and p17) and envelope (gp120, gp160 and gp41) proteins, into well-defined bands for use as HIV-1 antigen standards.  The separated bands are transferred or “blotted” onto a nitrocellulose membrane that is cut into strips and exposed to the serum sample.  Serum antibodies to the antigen standards are detected and characterised as discrete coloured bands by use of antihuman antibody in conjugation with an enzyme, as shown in Fig. 1.  The diagnostic pattern of bands identified in the Western blot is more specific than the ELISA for viral antibodies”.

An antibody test, Western blot (WB), cannot be used as a gold standard for another antibody test, ELISA.  Just because in the WB the “viral” antigens are separate, this is not proof that the WB is more specific than ELISA.    Neither can the specificity of an antibody test be determined by repeating the test, no matter how many times.  Furthermore, at present there is no proof that the “viral core (p24, p55 and p17) and envelope (gp120, gp160 and gp41) proteins” or any other protein used in the ELISA or WB are HIV proteins.9, 10
 
According to Luc Montagnier the characterisation of proteins as HIV proteins “demands mass production and purification [of the virus].  It is necessary to do that.  And there I should say that that partially failed”.11  In fact since the material which Montagnier et al used to characterise the “viral core” protein, p24, did not even have retrovirus-like particles, much less “purified” HIV, then one has no choice but to conclude that Montagnier and his associates did not prove that p24 is an HIV protein.  Neither has anybody else since.

When Djamel Tahi asked Montagnier if Robert Gallo purified HIV, he replied:  “Gallo ?…I don’t know if he really purified.   I don’t believe so”.  Like Montagnier, Robert Gallo and his colleagues did not publish electron micrographs to show that their “purified” virus contained retrovirus-like particles.  Unlike Montagnier el al who considered the protein of molecular weight 24,000 (p24) as being the characteristic HIV proteins, Gallo et al considered a protein of molecular weight 41,000 (p41), which is the molecular weight of actin, as the most specific HIV protein.  The only proof they gave for this was its banding at the density of 1.16gm/ml and reaction with the sera of AIDS patients.

In a Franco-German study, published  in 1997, the authors, which included Hans Gelderblom, pointed out that although the 1.16gm/ml band, which is used for “biochemical and serological analyses”, is “considered to contain a population of relatively pure virus particles,…in none of the studies has the purity of the virus preparation been verified”.5  However, by 1997, ample evidence existed which showed  that the 1.16gm/ml band contains many cellular proteins including actin and myosin, the latter also an ubiquitous  protein which has two light chains of molecular weight 24,000 and 18,000.  Evidence also exists that AIDS patients have antibodies to both actin and myosin.12

Before 1987 the p120 and p160 bands could not be visualised in WB strips.  This was not unexpected since according to the HIV experts p160 is present only in infected cells, not in virus particles, and p120 to be present only in the particles’ knobs (spikes), which are rapidly lost when the particles are released.  Since the protein on the WB strips are obtained from purified HIV particles which do not possess knobs13 210 (p120) then neither p120 nor p160 should be present.  Nevertheless, in 1987, by modifying “blot preparation”, proteins of molecular weights of 120,000 and 160,000 were found which reacted with patients sera.14 306  However, no amount of “blot preparation” modification can create what is not already present.  The explanation for the presence of these bands was found in 1989 by researchers who showed that in the Western blot strip, “the components visualised in the 120-160 kDa region do not correspond to gp120 or its precursor but rather represent oligomers of gp41”.15 248  It was also shown that the WB pattern obtained  is dependent on many factors including temperature and the concentration of sodium dodecyl sulphate used to disrupt the “pure virus”.  “Confusion over the identification of these bands has resulted in incorrect conclusions in experimental studies.  Similarly, some clinical specimens may have been identified erroneously as seropositive, on the assumption that these bands reflected specific reactivity against two distinct viral components and fulfilled a criterion for true or probable positivity.  The correct identification of these bands will affect the standards to be established for Western blot positivity:  it may necessitate the reinterpretation of published results”.16 773  No notice was taken of these findings and recommendations.

Definite proof that what is considered “purified” HIV, the 1.16 gm/ml band contains neither retroviral proteins nor HIV was published in 1997.  In that year, two papers were published in Virology with the first electron micrographs of “purified HIV” obtained by banding the supernatant of “infected” cultures in sucrose density gradients.  One of the studies was by researchers from the AIDS Vaccine Program SAIL, National Cancer Institute–Frederick Cancer Research and Development, Frederick, Maryland, USA and the other by researchers from France and Germany.4, 5  The authors of both studies claimed their “purified” material contain retrovirus-like particles and in fact that they were HIV particles.  But they admitted that their material predominantly contained particles which were not viruses but “mock virus”, that is “budding membrane particles frequently called microvesicles”.  Indeed, the caption to one of the electron micrographs of the “purified” HIV reads:  “Purified vesicles from infected H9 cells (a) and activated PBMC (b) supernatants".  It does not read “purified HIV”.  In further experiments the supernatants from non-infected cultures were also banded in sucrose gradients.  They claimed that the banded material from these cultures contained only microvesicles, “mock virus” particles, but no particles with the morphology of HIV.

No reason(s) is given, other than morphological, for why some of the particles in the fractions from the “infected” cells are virus particles and the others “mock virus”.  As far as morphology is concerned, none of the particles have all the morphological characteristics attributed to HIV, or even retroviruses.

The minimum absolutely necessary but not sufficient condition to claim that what are called “HIV-1 particles” are a retrovirus and not cellular microvesicles is to show that the sucrose density fractions obtained from the “infected” cells contain proteins which are not present in the same fractions obtained from non-infected cells that is in the “mock virus”.  However, the researchers from the USA have shown this is not the case.  The only difference one can see in their SDS-polyacrylamide gel electrophoresis strips of “purified virus” and “mock virus” is quantitative, not qualitative.  This quantitative difference may be due to many reasons including the fact that there were significant differences in the history and the mode of preparation of the non-infected and “infected” H9 cell cultures, in addition to the “infection”.  A similar finding was reported by the same authors a few years earlier.17    However, while in both studies the proteins of molecular weight “near 42 kDa” (42,000) are labelled as “Actin” and  “in the 30- to 40-kDa range” as “HLA DR”, all the proteins with molecular weight higher than approximately 42,000 and lower than approximately 30,000 are left unlabelled in the earlier paper.17  In the 1997 study, three proteins of molecular weight lower than 30.000 are labelled as p24CA, P17MA, and p6/p7NC and are said to represent “major bands of viral proteins”.  However, according to the authors, “these labels were added when one of the reviewers asked for them.  He felt if would help orient readers when looking at the figure–the reviewer is correct.  We did not determine the identities of the bands in the particular gel”.(Bess, personal communication).

Since both the “purified HIV” and the “mock” virus contain the same proteins, one has no choice but to conclude that the 1.16 g/ml band, the “purified HIV”:

(i)         has no HIV proteins and thus no HIV;

(ii)        the proteins used as antigens in both the ELISA and WB antibody tests are non HIV;

(iii)       since the only evidence which is said to prove that the antibodies present  in the AIDS patients sera are HIV antibodies is their reaction with the proteins which band at 1.16 gm/ml and the assumption that they are HIV;  and since no HIV proteins are present at this band;  it follows that the AIDS patients do not have HIV antibodies.

In conclusion, although evidence exists for a correlation between the antibody tests and AIDS, no evidence exists which proves that a positive antibody test means HIV infection.

REFERENCES

1.         Abbott Laboratories Diagnostics Division.  100 Abbott Park Rd. Abbott Park.  Illinois: USA.  1988, 1998.  Packet Insert Axsym system (HIV-1/HIV-2).

            http://aids-kritik.de/aids/diverses/abbott-hiv-test.htm

2.         WHO. HIV Test Kits.

3.         Sloand EM, Pitt E, Chiarello RJ, Nemo GJ. HIV Testing  State of the Art. Journal of the American Medical Association 1991; 266:2861-2866.

4.         Bess JW, Gorelick RJ, Bosche WJ, Henderson LE, Arthur LO. Microvesicles are a source of contaminating cellular proteins found in purified HIV-1 preparations. Virology 1997; 230:134-144.

5.         Gluschankof P, Mondor I, Gelderblom HR, Sattentau QJ. Cell membrane vesicles are a major contaminant of gradient-enriched human immunodeficiency virus type-1 preparations. Virology 1997; 230:125-133.

6.         Schupbach J, Jendis JB, Bron C, Boni J, Tomasik Z. False-positive HIV-1 virus cultures using whole blood. AIDS 1992; 6:1545-6.

7.         Mortimer P, Codd A, Connolly J, et al. Towards error free HIV diagnosis: notes on laboratory practice. Public Health Laboratory Service Microbiology Digest 1992; 9:61-64.

8.         WHO. HIV type 1 variation in World Health Organization-sponsored vaccine evaluation sites: genetic screening, sequence analysis, and preliminary biological characterization of selected viral strains. AIDS Research and Human Retroviruses 1994; 10:1327-1343.

9.         Papadopulos-Eleopulos E, Turner VF, Papadimitriou JM, Causer D. The Isolation of HIV: Has it  really been achieved? Continuum 1996; 4:1s-24s.

10.       Papadopulos-Eleopulos E, Turner VF, Papadimitriou JM. Has Gallo proven the role of HIV in AIDS? Emergency Medicine [Australia] 1993; 5:113-123.

11.       Tahi D. Did Luc Montagnier discover HIV?  Text of video interview with Professor Luc Montagnier at the Pasteur Institute July 18th 1997. Continuum 1998; 5:30-34.

12.       Matsiota P, Chamaret S, Montagnier L. Detection of Natural Autoantibodies in the serum of Anti-HIV Positive-Individuals. Annales de l'Institut Pasteur Immunologie 1987; 138:223-233.

13.       Layne SP, Merges MJ, Dembo M, et al. Factors underlying spontaneous inactivation and susceptibility to neutralization of human immunodeficiency virus. Virology 1992; 189:695-714.

14.       Burke DS. Laboratory diagnosis of human immunodeficiency virus infection. Clinical and Laboratory Medicine 1989; 9:369-392.

15.       Pinter A, Honnen WJ, Tilley SA, et al. Oligomeric structure of gp41, the transmembrane protein of human immunodeficiency virus type 1. Journal of Virology 1989; 63:2674-9.

16.       Zolla-Pazner S, Gorny MK, Honnen WJ. Reinterpretation of human immunodeficiency virus Western blot patterns. New England Journal of Medicine 1989; 320:1280-1281.

17.       Arthur LO, Bess JW, Jr., Urban RG, et al. Macaques immunized with HLA-DR are protected from challenge with simian immunodeficiency virus. Journal of Virology 1995; 69:3117-24.
« Τελευταία τροποποίηση: Οκτώβριος 10, 2019, 23:07:35 από Rose »
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Απ: HIV virus does not cause AIDS Acquired Immune Deficiency Syndrome
« Απάντηση #40 στις: Οκτώβριος 10, 2019, 23:21:43 »
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Απ: HIV virus does not cause AIDS Acquired Immune Deficiency Syndrome
« Απάντηση #42 στις: Οκτώβριος 10, 2019, 23:27:54 »
Peter Duesberg, PhD
https://www.youtube.com/watch?time_continue=118&v=DOMov4Wot60

Peter Duesberg, PhD, presents "the HIV-AIDS Hypothesis-30 Years Later" at the 2015 IAOMT Annual Conference in Las Vegas, Nevada.
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Απ: HIV virus does not cause AIDS Acquired Immune Deficiency Syndrome
« Απάντηση #43 στις: Οκτώβριος 10, 2019, 23:41:22 »
Living Without HIV Drugs
http://livingwithouthivdrugs.com/

Living without HIV drugs
This website is dedicated to the true-life stories of people who have two important things in common:

1. We have all been diagnosed as HIV-Positive at some point in our lives, and
2. We have all stopped, or never started taking the HIV medications normally prescribed by our doctors and clinics and hospitals, and are living healthy, happy lives.
We want to share our stories with you, in hopes that they can be an inspiration and provide you with the hope that you, too, can live without HIV drugs and their very serious and damaging side effects.

Keep in mind that the HIV medications that you may be taking now (or will probably be pressured into taking if you are newly diagnosed as HIV-Positive) have been proven to be more dangerous than HIV itself. According to a presentation at the AIDS Conference in Barcelona in 2002, and a scientific study published in the Journal of Acquired Immune Deficiency Syndrome (December, 2003), more HIV-Positives are dying from organ failure as a side effect from the HIV drugs than are dying from AIDS; and according to the largest and longest scientific study of HAART (Highly Active Anti-Retroviral Therapy), the drugs being given today are even more dangerous and offer no increase in longevity than those given in 1996.

In the lefthand column is a list of names (in alphabetical order) of those who want to share their stories with you.  Simply click on a name to read their story. Some of them also have an email address at the bottom of the story if you would like to communicate with them directly, and some of them are available to listen to as podcasts by clicking here.

In the righthand column are links to other important HIV/AIDS websites, including two free ebooks.

If you would like to share your story with us (and others who might be in your same or similar situation), please click here.
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Απ: HIV virus does not cause AIDS Acquired Immune Deficiency Syndrome
« Απάντηση #44 στις: Οκτώβριος 11, 2019, 00:10:32 »
Infectious AIDS is a collection of thirteen articles originally published in scientific journals, which call into question the dogma of infectious AIDS. With such thought-provoking chapters as "HIV is Not the Cause of AIDS" and "AIDS acquired by Drug Consumption and Other Non-contagious Risk Factors," Duesberg explores the correlation (but not causality) between HIV and AIDS. By challenging popular AIDS theory, Duesberg investigates fresh possibilities that can transform the study and treatment of the AIDS virus.

Infectious AIDS: Have We Been Misled?: The Fallacy of the HIV-AIDS Connection Paperback – December 13, 1995

https://www.amazon.com/Infectious-AIDS-Fallacy-HIV-AIDS-Connection/dp/1556431953/ref=pd_sbs_14_t_2/142-5249496-5506445?_encoding=UTF8&pd_rd_i=1556431953&pd_rd_r=5501b558-2a50-4e96-82a8-ca8eb67aab42&pd_rd_w=s93mU&pd_rd_wg=EtRVF&pf_rd_p=5cfcfe89-300f-47d2-b1ad-a4e27203a02a&pf_rd_r=F6CD95FCTHANVJWBD72C&psc=1&refRID=F6CD95FCTHANVJWBD72C
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